Term
| Parathyroid hormone (PTH) |
|
Definition
| synthesized and secreted by the chief cells of the parathyroid glands. |
|
|
Term
|
Definition
| located in the outer rim of islet, secrete glucagon |
|
|
Term
|
Definition
| located in the central part of the islet, secrete insulin. |
|
|
Term
|
Definition
| are intermixed and secrete somatostatin and gastrin. |
|
|
Term
|
Definition
increases the blood glucose concentration.
It increases glycogenolysis and prevents recycling of glucose into glycogen
It increases gluconeogenesis
It increases blood fatty acid and ketoacid concentration.
It increases lipolysis,
Glucagon increases urea Production |
|
|
Term
|
Definition
| increases glucose uptake, promotes glycogenesis, decreases gluconeogenesis, decreases blood fatty acid and ketoacid concentrations, insulin decreases blood K+ concentration. |
|
|
Term
|
Definition
| Effect – prolong digestive window for nutrient assimilation into blood and decreases nutrient utilization |
|
|
Term
|
Definition
| Day/Night, Sleep cycles, Melatonin. |
|
|
Term
|
Definition
| Estrogen, Progesterone , Testosterone, hCG |
|
|
Term
|
Definition
arachidonic acid is converted by the enzyme cyclooxygenase into a prostaglandin.
-PGI2, antiplatelet aggregation, vasodilation -PGE2, smooth muscle relaxation, vasodilation -PGF2alpha, smooth muscle contraction, vasoconstriction -TXA2 (thromboxane A2), platelet aggregation, vasoconstriction |
|
|
Term
|
Definition
| Arachidonic acid is converted by the enzyme lipoxygenase into leukotrienes which are largely responsible for the symptoms of asthma. -Inflammation -Bronchoconstriction -Vasoconstriction |
|
|
Term
| Inhibitors of Prostaglandins synthesis |
|
Definition
| Nonsteroidal anti-inflammatory drugs (NSAIDs) Indomethacin Ibuprofen These drugs produce their effects because they specifically inhibit the cyclooxygenase enzyme that is needed for prostaglandin synthesis. |
|
|
