CHARACTERIZED BY A DECLINE FROM A PREVIOUSLY ATTAINED LEVEL OF FUNCTIONING

-MEMORY IMPAIRMENT

-MUST HAVE ONE OR MORE OF:

APHASIA

APRAXIA

AGNOSIA

OF CHANGES IN EXECUTIVE FUNCTIONING

CATEOGRY 1

-RELATED TO THE BRAIN

CATEGORY 2

-PSYCHOLOGICAL

*MANY DISORDERS ARE CATEGORY 1 AND 2 (BRAIN AND PSYCHOLOGICAL)

CATEGORY 1 AND 2 WERE REPLACED BY:

DELERIUM

DEMENTIA

AMNESTIC SYNDROME

LOSS OF FUNCTIONING

SEVERITY DEPENDS ON NATURE, LOCATION, EXTENT OF DAMAGE, COMPETENCY OF PERSON BEFORE THE ACCIDENT, LIFE SITUATION, AND AMOUNT OF TIME SINCE BRAIN DAMAGE FIRST OCCURED

DELIRIUM

DEMENTIA

AMNESTIC SYNDROME

-COMMONLY OCCURING

ACUTE CONFUSIONAL STATE THAT IS SOMEWHERE BETWEEN NORMAL WAKEFULNESS AND STUPOR OR COMA

-INFORMATION PROCESSING IS AFFECTED

ATTENTION

PERCEPTION

MEMORY

THINKING

*CAN OCCUR IN ANY AGE, MOST COMMIN IN THE ELDERLY AND CHILDREN

DRUG INTOXICATION OR WITHDRAWAL( MOST COMMON)

HEAD INJURY

INFECTION

OFTEN REVERSIBLE WITH RIGHT TREATMENT UNLESS CAUSED BY TERMINAL ILLNESS OR SEVERE BRAIN TRAUMA

TREATED WITH MEDICATIONS

ENVIRONMENTAL MANIPULATIONS

FAMILY SUPPORT

OVER 50 DIFFERENT DISORDERS CAN CAUSE IT

-ALZHEIMERS (DEGENERATIVE)

-PARKINSONS (DEGENERATIVE)

-HUNTINGTONS (DEGENERATIVE)

-STROKE

-INFECTIOUS DISEASE (HIV)

-DIETARY DEFICIENCIES (VIT. B)

-HEAD INJURY

-INGESTION OF MERCURY OR LEAD

AGE (DOUBLES EVERY 5 YEARS AFTER 40)

GENDER (WOMEN ARE MORE LIKELY)

GENETIC MUTATION S SPECIFIC GENES

CELL LOSS (PLAQUES AND NEUROFIBRILLARY TANGLES)

SECOND MOST COMMON NEURODEGENERATIVE DISORDER

MORE COMMON IN MEN THAN WOMEN

AGE 50-70

RARE

HEREDITARY

CAN BE TESTED FOR BUT MANY PEOPLE DON'T WANT TO KNOW

=IN MEN AND WOMEN

DESTRUCTION OF BRAIN CELLS

MILD MEMORY DIFFICULTIES

PSYCHOMOTOR SLOWING

DIMINISHED ATTENTION AND CONCENTRATION

*20-30% OF PEOPLE WITH ADVANCED HIV HAVE AIDS-RELATED DEMENTIA

WEAKENS THE IMMUNE SYSTEM OR EFFECTS THE BRAIN DIRECTLY

LOOKS LIKE ALZHEIMERS, OFTEN MISDIAGNOSED

CAUSED BY INTERRUPTIONS TO THE BLOOD SUPPLY TO THE BRAIN DUSE TO SMALL STROKES

OVER TIME LEADS TO HOLES IN THE BRAIN

NEW MEDICAL TREATMENTS HAVE BEEN DEVELOPED TO ATTEMPT TO STOP THE STROKES

DISTURBED MEMORY

-MAY REMEMBER ONLY THE PAST OR ONLY EVENTS AFTER THE EVENT OR ONLY A FEW MINS OF INFO

IMPAIRMENT IN FUNCTIONING

MEMORY LOSS IS EVIDENT NOT DUE TO DELIRIUM OR DEMENTIA

CHRONIC ALCOHOL USE

HEAD TRAUMA

STROKE

CLOSED HEAD INJURY

PENETRATING HEAD INJURY (BULLET)

SKULL FRACTURE (FALL, ACCIDENT)

*MEN 15-24

RETROGRADE AMNESIA (BEFORE EVENT)

ANTEROGRADE AMNESIA (AFTER EVENT)

PERSONALITY CHANGES

*PHINEAS GAGE

IMMEDIATE TREATMENT(RELEASE PRESSURE ON THE BRAIN, REMOVE OBJECT)

FAMILY THERAPY

RELEARNING SKILLS THROUGH OCCUPATIONAL THERAPY

FOR MANY THE SYMPTOMS DISSIPATE

MANY HAVE LIFE LONG SYMPTOMS

-HEADCHES

-NAUSEA

-DIZZINESS

-TEMPER CHANGES, PERSONALITY CHANGES

-IMPAIRED MEMORY, CONCENTRATION

-INCREASED RISK OF DEPRESSION, ANGER, ANXIETY DISORDERS

SCHIZOPHRENICS HAVE 2 OR MORE OF?

-DELUSIONS

-HALLUCINATIONS

-DISORGANIZED SPEECH

-GROSSLY DISORGANIZED OR CATATONIC BEHAVIOR

-NEGATIVE SYMPTOMS

AND THEY ALWAYS HAVE DYSFUNCTION AT WORK, N RELATIONSHIPS, AND CARING FOR SELF

ADDITION OF A BEHAVIOR, THOUGHT

-DELUSIONS 90+%

-HALLUCINATIONS (AUDITORY 75%, VISUAL 15%) SENSORY EXPERIENCE I THE ABSENCE OF AN EXTERNAL STIMULUS

-DISORGANIZED SPEECH (SENTENCES THAT ARE GRAMMATICALLY CORRECT, BUT INCOHERENT)

-DISORGANIZED BEHAVIOR (STOP BEHAVING IN SOCIALLY APPROPRIATE WAYS)

-CATATONIA (ABSENCE OF MOVEMENT, AND SPEECH-HOLD ONE POSITION FOR A VERY LONG TIME)

NEGATIVE SYMPTOMS REFLECT AN ABSENCE OR DEFICIT OF BEHAVIORS THAT ARE NORMALLY PRESENT

-FLAT OR BLUNTED EMOTIONAL EXPRESSIVENESS

-ALOGIA

-AVOLITION

-OFTEN SOMEONE WILL JUST SIT ALL DAY STARING

-PARANOID TYPE (SPECIFIC)

-DISORGANIZED TYPE (SPECIFIC)

-CATATONIC TYPE (SPECIFIC)

-UNDIFFERENTIATED TYPE (NOT SURE)

-RESIDUAL TYPE (NOT SURE)

HISPTORY OF SUSPICIOUSNESS, DIFFICULTY IN INTERPERSONAL RELATIONSHIPS

DELUSIONS OF PERSECUTION AND GRANDEUR- SOMEONE IS AFTER THEM AND BELIEVING THEY ARE SOMEONE THEY AREN'T

HIGH FUNCTIONING, INTELLIGENCE THAN OTHER SUBTYPES

HIGHEST TREATMENT SUCCESS

EARLY, GRADUAL ONSET 15-18

DISORGANIZED SPEECH

DISORGANIZED BEHAVIOR

FLAT AFFECT, INAPPROPRIATE AFFECT

TREATMENT HAS NOT BEEN FOUND

CATATONIC TYPE

2 OF THE FOLLOWING:

-IMMOBILE BODY

-EXCESSIVE MOTOR ACTIVITY

-EXTREME NEGATIVISM (RESISTANCE TO BEING MOVED, FED, ECT) OR MUTISM

-HOLD WEIRD POSTURES FOR LONG PERIODS

**-ECHOLALIA (IMITATE OTHERS WORDS)

**-ECHOPRAXIA (IMITATE OTHERS ACTIONS

MAY CYCLE THROUGH CATATONIC STATE TO EXTREME ACTION PHASE CAN BE DANGEROUS TO SELF AND OTHERS

NOT AS PREVALENT IN US TODAY AS IN THE PAST BECAUSE THE PROGNOSIS IS POOR

DIAGNOSED AS SCHIZOPHRENIC

SYMPTOMS DO NOT FIT A SPECIFIC TYPE

SUFFERED AT LEAST ONE EPISODE OF SCHIZOPHRENIC BEHAVIOR

TEND TO HAVE NEGATIVE SYMPTOMS, WITHOUT POSITIVE SYMPTOMS OR THE POSITIVE ONES ARE MILD

GENETIC FACTORS

HAVING A RELATIVE WITH THE DISORDER SIGNIFICANTLY RAISES A PERSON'S RISK OF DEVELOPING SCHIZOPHRENIA

HAVING A FATHER WHO WAS OVER 45 AT THE TIME OF CONCEPTION

PRENATAL EXPOSURE TO THE INFLUENZA VIRUS

EARLY NUTRITIONAL DEFICEINCIES

PRENATAL BIRTH COMPLICATIONS (BREACH)

LESION I NTHE BRAIN IS THOUGHT TO LIE DORMANT UNTIL NORMAL DEVELOPMENTAL CHANGES OCCUR

THESE CHANGES EXPOSE THE PROBLEMS THAT RESULT FROM THIS BRAIN ABNORMALITY

DECREASED BRAIN VOLUME

ENLARGED VENTRICLES

FRONTAL LOBE DYSFUNCTION

REDUCED VOLUME OF THE THALAMUS

ABNORMALITIES IN TEMPORAL LOBE AREAS SUCH AS THE HIPPOCAMPUS AND AMYGDALA

-THESE ABNORMALITIES ARE NOT FOUND IN ALL PATIENTS

SAYS THAT IT IS CAUSED BY HAVING A COLD MOTHER THAT DIDN'T GIVE ATTENTION.

DISPROVED

THOSE WITH SCHIZOPHRENIA WHO LIVE WITH FAMILY AFTER TREATMENT HAVE AN INCREASE IN RELAPSE VERSUS THOSE WHO LIVE ALONE

EXPRESSED EMOTION LEVEL OF THE FAMILY PREDICTS RELAPSE

LIVING IN AN URBAN ENVIRONMENT=HIGH LIKELIHOOD OF SCHIZOPHRENIA

AFTER TREATMENT OVER 35% HAVE A GOOD OUTCOME--THEY ARE RECOVERED BUT STILL NEED TREATTMENT

OVER 15% NO LONGER NEED TREATMENT

12% NEED TO BE INSTITUTIONALIEZD PERMANETNLY

OVER 30% CONTINUE TO HAVE NEGATIVE SYMPTOMS

ANTIPSYCHOTIC DRUGS

-HELP WITH POSITIVE SYMPTOMS

-SIDE EFFECTS INCLUDE MUSCLE SPASMS, AND SHAKING

THE SIDE EFFECTS ARE REDUCED IN NEWER SECOND GENERATION ANTIPSYCHOTICS

-2ND GENERATION SIDE EFFECTS IN CLUDE DROWSINESS AND WEIGHT GAIN

PSYCHOSOCIAL APPROACHES

-CASE MANAGEMENT (PATIENTS REPRESENTATIVE)

-SOCIAL-SKILLS/VOCATIONAL TRAINING (TEACH BASIC SOCIAL SKILLS)

-COGNITIVE-BEHAVIORAL THERAPY (FOCUS ON TEACHCING PATIENT TO THINK CIRITICALLY ABOUT THEIR POSITIVE SYMPTOMS)

-PERSONAL THERAPY (TEACHES COPING SKILLS)

-FAMILY THERAPY (PROVIDES FAMILIES WITH COMMUNICATION SKILLS, REDUCES HIGH LEVELS OF EXPRESSED EMOTION)

A LOT OF RESEARCH BEING DONE

THOSE WHO ARE AT HIGH RISK CAN BE PUT ON LOW DOSE ANTIPHYCHOTICS AND DO FAMILY THERAPY AND CBT WITH THEM

*RELATIONSHIP BETWEEN CLIENT AND CLINICIAN IS IMPORTANT.

THE PATIENT MUST TRUST THEM

THEY DO NOT HAVE FULLY DEVELOPED COPING SKILLS

THEY HAVE LESS SELF-UNDERSTANDING

THEY DO NOT UNDERSTAND LIFE EVENTS AS WELL

THEY ARE MORE DEPENDENT ON OTHERS

IMPUSIVITY

EXAGGERATED MOTOR ACTIVIY

CONSTANT TALKING

LACK OF SUSTAINED ATTENTION

LOWERED INTELLIGENCE

IMMATURITY

**THE MOST COMMON REASON FOR A CHILD TO GET A PSYCHOLOGICAL REFERRAL

**MOST COMMON IN BOYS

RARELY CONTINUES INTO ADOLESCENCE AND ADULTHOOD

SOME DON'T BELIEVE IT EVEN EXISTS IN ADULTS

MAY BE CAUSED BY ENVIRONMENT AND GENETICS

**RITALIN

COGNITIVE BEHAVIORAL METHODS

-SELECTIVE REINFORCEMENT

FAMILY THERAPY

OPPOSITIONAL DEFIANT DISORDER CAN LEAD TO CONDUCT DISORDER WHICH IS:

BIOLOGICAL FACTORS

-TEMPERMENT AND INTELLIGENCE

SOCIAL INFLUENCES

-SES

-FAMILY PATTERS (DISTANT PARRENTS, REJECTION, INCONSISTENT PARENTING, NEGLECT, PARENT OFTEN HAS A DISORDER THEMSELVES

-PEER RELATIONSHIPS (BEHAVIOR LEADS TO PEER REFECTION, WHICH LEADS TO MORE POOR BEHAVIOR)

JAIL OR JUNENILE DETENTION (PUNITIVE) MAKES BEHAVIOR WORSE

EFFECTIVE TREATMENT FOCUS ON THE COHESIVE FAMILY MODEL (TEACH PARENTS HOW TO BE GOOD PARENT ROLE MODEL, SOMETIMES HAVE TO REMOVE CHILD FROM HOME--CAN HAVE NEGATIVE IMPACT)

BEHAVIORAL TECHNIQUES (TAUGHT TO PARENTS TO USE ON THEIR CHILDRE)

-UNREALISTIC FEARS

-OVERSENSITIVITY

-SELF-CONSCIOUSNESS

-NIGHTMARES

-CHRONIC ANXIETY

THE PERSISTENT FAILURE TO SPEAK IN SPECIFIC SOCIAL SITUATIONS

OFTEN RELATED TO DEVELOPMENTAL DELAY

ONLY DIAGNOSED IF THE CHILD CAN SPEAK, AND HASN'T IN A SPECIFIC SITUATION FOR MORE THAN A MONTH, NOT THE FIRST MONTH OF SCHOOL

FAMILY THERAPY

SOMETIMES ANTI-ANXIETY MEDS

EARLY FAMILY RELATIONSHIPS THAT GENERATE ANXIETY AND PREVENT THE CHILD FROM DEVELOPING MORE ADAPTIVE COPING SKILLS

HIGH LEVEL OF SENSITIVITY

OVERANXIOUS PARENTS

DETACHED PARENTS

PSYCHOPHARMACOLOGICAL TREATMENS AND BEHAVIOR THERAPY ARE COMMON

OFTEN THEY WORK THEMSELVES OUT BEFORE ADOLESCENCE OR ADULTHOOD

MORE IN ADOLESCENSE (15-20%) THAN IN CHILDREN (4-8%)

TWO TIMES MORE LIKELY IN GIRLS THAN BOYS

INCREASES SUICIDE ATTEMPT

BIOLOGICAL FACTORS

-MOOD DISORDERED FAMILIES

-0PRENATAL EXPOSURE TO ALCOHOL

LEARNING FACTORS

-FROM PARENTS AND FROM CULTURE

MEDICATION

-USUALLY ONLY ON ADULTS

TEACHING PARENTS HOW TO DEVELOP AN EMOPTIONALLY SUPPORTIVE ENVIRONMENT

TEACHING ADAPTING AND COPING SKILLS

CBT

GROUP OF SEVERLY DISABLING CONDITIONS

-THE MOST DIFFICULT TO UNDERSTAND AND TREAT

-DUE TO BRAIN DEFECTS FROM BIRTH

-CARRY ON THROUGH LIFE

-VARYING DEGREES OF SEVERITY WITHIN EACH DISORDER

SEVERE AND SUSTAINED IMPAIRMENT IN SOCIAL INTERACTION

RIGIDLY STICK TO ROUTINE

INFLEXIBILITY

AFFECTS 30-60 OF EVERY 10,000 CHILDREN

AGE OF DIAGNOSIS IS USAULLY AROUND 2-2.5

EVEN AS BABIES SEEN AS ALOOP, UNINTERESTED IN HUMAN CONTACT

SOCIAL DEFICIT (DON'T WANT SOCIAL CONTACT, LACK OF INTEREST IN PARENTS)

ABSENCE OF SPEECH (MAY ENGAGE IN ECHOLALIA)

SELF-STIMULATION (HEAD-BANGING, SPINNING, ROCKING)

IMPAIRED INTELLECTUAL ABILITY (MEMORY, SOCIAL REASONING)

PREOCCUPTAION WITH MAINTAINING SAMENESS (CARRY OBJECT AROUND, NEED ENCIORNMENT TO BE THE SAME AT ALL TIMES)

AUTISM CONT.

TJE CAUSES ARE UNKNOWN

MANY FUNDAMENTAL DISTURBANCES OF THE CENTRAL NERVOUS SYSTEM ARE INVOLVED

HIGHLY GENETIC RISK FACTOR-MORE THAN MOST OTHER DISORDERS

MANY BELIEVE IT IS RELATED TO VACCINES

DIFFICULT

NO KNOW TREATMENT OR CURE

SOME TREATMENTS MAY INCREASE FUNCTIONING (BEHAVIOR TRAINING--TAKES YEARS, OFTEN DONE BY PARENTS WHO ARE TRAINED)

CONTROVERSIAL BELIEF---DEFEAT AUTISM NOW, SLOGAN AUTISM IS TREATABLE

RESTRICTED TO THOSE CASES IN WHICH:

-THERE IS CLEAR IMPAINMENT IN SCHOOL PERFORMANCE OR IN DAILY LIVING ACTIVITIES

-THE IMPAIRMENT IS NOT DUE TO MENTAL RETARDATION OR TO A PERVASIE DEVELOPMENTAL DISORDER SUCH AS AUTISM

ARE POSSIBLY THE PRODUCTS OF SUBTLE CENTRAL NERVOUS SYSTEM IMPAIRMENTS

MAY BE ENVIORONMENTAL

TREATMENT IS NOT WELL RESERACHED

DISABILTY SUPPORT SERVICES- HELP CHILDREN OVERCOME THEIR DISABLITY

SIGNIFICANTLY SUBAVERAGE GENERAL INTELLECTUAL FUNCTIONING....THAT IS ACCOMPANIED BY SIGNIFICANT LIMITATIONS IN ADAPTIVE FUNCTIONING.

MUST BE DIAGNOSED BEFORE AGE 17, OTHERWISE CONSIDERED DEMENTIA

MILD MENTAL RETARDATION

MODERTE MENTAL RETARDATION

SEVER MENTAL RETARDATION

PROFOUND MENTAL RETARDATION

MILD RETARDATION

IQ FROM 50-55 TO 70

LARGEST GROUP

CONSIDERED "EDUCABLE"

AS AN ADULT

INTELECTUALLY SIMILAR TO AN 8-11 YEAR OLD, BUT WITH MORE LIFE EXPERIENCES

SOCIALLY CONSIDERED SIMILAR TO AN ADOLESCENT WITH LESS IMAGINATION, AND JUDGEMENT

DIAGNOSIS

-WITH PARENTAL HELP AND EDUCATION CAN PROGRESS WELL IN SOCIETY, BE SELF-SUPPORTING AS ADULTS

MODERATE MENTAL RETARDATION

IQ FROM 35-40 TO 50-55

CONSIDERED TRAINABLE

CAN LEARN HOW TO DO BASIC SKILLS, LIKE COOK

INTELLECTUALLY THE SAME AS 4-7 YEAR OLD

SLOW LEARNERS, BODY DEFORMITIES, IMPAIRMENTS TO MOTOR FUNCTIONING

DIAGNOSIS

-WITH PARENTAL SUPPORT, TRAINING, CAN BE PARTIALLY INDEPENDENT

SEVERE MENTAL RETARDATION

IQ 20-25 TO 35-40

CONSIDERED DEPENDENT

SEVERE IMPAIRMENT IN MOTOR AND SPEECH DEVELOPMENT

SENSORY DEFICITS

DIAGNOSIS

-WITH PARENTAL SUPPORT, AND TRAINING CAN ACHIEVE SIMPLE OCCUPATION TASKS UNDER SUPERVISION

PROFOUND MENTAL RETARDATION

IQ BELOW 20-25

CONSIDERED LIFE SUPPORTED RETARDED

CONVULSIVE SEIZURES

MUTISM, DEAFNESS

DIAGNOSIS

-CARED FOR FULL TIME FOR LIFE

-POOR HEALTH, SHORT LIFE EXPECTANCY

GENETIC-CHROMOSOMAL FACTORS

-EXTRA CHROMOSOME 21=DOWN SYNDROME

INFECTIONS AND TOXIC AGENTS

-GENITAL HERPES WHILE PREGO

-DRUG ALCOHOL USE DURING PREGO

TRAUMA (PHYSICAL INJURY)

-TRAUMA DURING BIRTH--BLEEDING, HYPOXIA

IONIZING RADIATION

-X-RAYS

MALNUTRITION

-LACK OF PROTEIN, CERTAIN VITAMINS DURING PREGO

TYPE OF MENTAL RETARDATION CAUSED BY GENETIC ABNORMALITY

3 CHROMOSOME 21--47 CHROMOSOMES

BODY FEATURES ARE DIFFERENT, OFTEN OBVIOUS A PERSON HAS DOWN SYNDROME FROM THEIR PHYSICAL APPEARANCE

ACCELERATED AGING

VERBAL AND LANGUAGE SKILL IMPAIRMENT

VARYING DEGREES OF SEVERITY

RISK FACTORS

-MOTHER AND FATHER AGE

INSTITUTIONALIZATION (USUALLY A LAST RESORT)

EDUCATION

MAINSTREAMING

NOW OVER 5 MILLION PEOPLE

IN 50 YEARS THERE WILL BE 14 MILLION

RISK FACTORS:

GREATEST IS AGE 65 AND UP, ONCE 85 THE RISK FACTOR RISES TO 50%

FAMILY HISTORY

HEAD INJURY

HIGH BLOOD PRESSURE

HIGH CHOLESTEROL

HEAR DISEASE

STROKE

OUT OF THE BLUE THEY NOTICED AN ODD SENSATION IN THEIR LEG.  FINER WOULD TREMBLE.  LEFT AND RIGHT ARMS WERE SWINGING DIFFERENTLY.  SHIRT MORE DIFFICULT TO BUTTON, NECLACE MORE DIFFICULT FO CLASP.

SLOW TICKING DISEASE

PHONE VIBRATING IN POCKET BUT NO PHONE THERE

CURES?

EMBRYONIC STEM CELL RESEARCH

CLIVE

MEMORY IS 30 SECS AND SOMETIMES ONLY 7 SECS.

CAN STILL PLAY THE PIANO

STILL KISSES

STILL EATS

STILL OPENS THE DOOR FOR HIS WIFE

STILL SHOWS EMOTIONS

STILL KNOW HOW TO DANCE

WORST CASE OF AMNESIA EVER KNOWN

WIFE IS ONLY PERSON HE RECOGNIZED

EVERY TIME HE SEES HER IT'S LIKE THE FIRST TIME

ALZHEIMER'S IS THE 8TH LEADING CAUSE OF DEATH

220% RISE IN 10 YEARS

4 MILLION WILL HAVE IT

10 WARNING SIGNS

51% OF CAREGIVERS ARE TREATED FOR DEPRESSION

31%WOULD CONSIDER THEIR HEALTH POOR

WAYNE KEEFER

POWERFUL VOICES

BRAIN DISEASE

CHRONIC, LONG-TERM DISEASE

STARTED BVY HEARING VOICES OUT OF HIS DOOR

18-19 ONSET FOR HIM

MUTATIONS ON CHROMOSOME 6

GENETICS IS A MAJOR RISK FACTOR

GENTLEST PEOPLE

USUALLY NO RISK OF ANGER AND/OR PHYSICL VIOLENCE

1ST GENERATION- (TYPICAL)NEUROTRANSMITTER BLOCKING. LOTS OF SIDE EFFECTS

2ND GENERATION-(ATYPICAL) SEROTONIN AND DOPAMINE. LESS SIDE EFFECTS AND RELIEVE MORE SYMPTOMS

AKATISIA(FEELING OF RESTLESSNESS)

75% OF SCHIZOPHRENICS RECOVER WITH MEDICATION AND THERAPY

ANTIPSYCHOTICS-ALLEVIATE CHEMICAL IMBALANCES IN THE BRAIN

REHABILITATINO THERAPY

FAMILY THERPY SENSE OF PRODUCTIVITY

CBT

COPING MECHANISMS TO ALLEVIATE SYMPTOMS AND PREVENT RELAPSE

SUBSTNACE ABUSE TREATMENT

ESCAPE FROM VOICES

NEARLY 50% OF SCHIZO ABUSE DRUGS

SOCIAL AND VOCATIONAL REHABILITATION

CAREER TRAINING

SOCIAL AND COMMUNICATION SKILLS

FAMILY EDUCATION THERAPY

FAMILY SUPPORT SERVES AS AN ANCHOR

MUST BE DIAGNOSED BEFORE 7

BEHAVIORAL DISORDER

FIRST IDETIFIED IN 1902

SIZE AND DENSITY OF BRAIN

-SMALLER CORTEX

FRONTAL LOBE-SMALLER NERVE TISSUE

CHEMICAL DIFFERENCE

-LOWER DOPAMINE

PSYCHOSTIMULANTS

SNRIS