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| How do you determine plasma osmolarity |
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| prcessed canned foods slty often hidden in medicines (alkiseltzer) |
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| decreased Na could be caused by.. |
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| prolonged administration of hypotonic solutions. or decreased sodium intake over several months |
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| peripheral edema, wt gain.pulm edema |
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| Intraventions for fluid overload |
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| monitor I/O with restricted fluids, daily weights labs, give diuretic, encourage eating salty foods |
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| prolongd administraton of hypertonic soulutions |
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| signs and symptoms of increased Na |
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| dry mucous membranes and skin . wt. loss, inreased rr low grade fever poor skin truger, sunken eyebaalls confusion seizures increae urine consentraton |
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| interventions for increased Na |
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| could igve .45% hypotonic solution but worry about giving too mcuh and going into FVE May also see LR given if cardiac and renal system can handle it. this will correct fvd and in turn Na concentration. |
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| an intracellular electrolyte |
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| K+ maintains action potential in |
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| muscles , neurons, and other tissues |
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| K+ assisits in contolling |
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| 90% kidneys and a little through stool and perspiration |
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green leafy, banana's, cantaloups, peaches, oranges, milk, nuts salt substitutes are high in K |
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| altered cell function (trauma to cell, metabolic acidosis, blood transfusion with ould blood. |
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| Signs and symptoms of low K |
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| irregulare andslow HR, EKG cahnges (peaked T waves) muscle cramps (especially in abdominal) |
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cardiac monitor administor antidote -kayexalate -calcium gluconate -IV insulin monior labs assess for chest pain |
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| P- and what are used interchangeabley in the medical field |
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| P- acts in a seesaw effect with |
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| dairy (milk cheese yogurt) |
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Vit D is needed for absorption, present in bone and teeth, stored in bone and and excreted in the kidney -clotting-it converts prothrombin inot thrombin for clot formation -it also has a role in bone and teeth formation |
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| dariy, salmon , green veggies, baked beans |
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| what is responsible for regulation of Ca+? |
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| What happens when PTH is released? |
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| it causes Ca+ to move out of the bone into the blood to correct any low extracellular Ca+ levels b/c Ca is always inversly rlated to P- and drives P- out of the blood |
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| a hormone from the thyroid that does the opposite of PTH. It increases calcium return to the bone, takin git from the blood |
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| Increased Ca+ can be caused by? |
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| reanl failure, use of thiazide diuretics, prolonged immobility, hyperparthyroidisn/ bone cancer or decreased P- |
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| depression, flabby muscles, EKG changes bone fractures, kidney stones |
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| Interventions of high Ca+ |
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promote activity maintain hydration administer lasix as ordered |
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| Low Ca+ can be caused by ... |
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| decreaded vid D increased P- hypoparathyroidism |
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| tetany (tight twitching muscle movements)abdominal cramps, Chvostek and Trousseau if severe EKG changes (weak contrations of the heart) clotting abnormality |
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neuromuscular, cardiac, and enzyme activity, helps maintain muscle activity so without enough tetany like problems or with too much slowed responses -it acts a lot like Ca+ and K+ in its roles and Mg imbalances are often accompaniend by a Ca+ or K+ imbalance |
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| lethargy, weakness, loss of deep tendon reflexes, hypotention, repiratory depression |
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| hyperirritability/ increaed deep tendon reflexes, HTN, dysrhythmias, tetany-like symptoms |
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