Term
|
Definition
| first generation h1 blocker. Mechanism: reversible inhibition of histamine receptors. uses: allergy, motion sickness, sleep aid. tx: sedation, antimuscarinic (hence the sleepy), motion sickness |
|
|
Term
|
Definition
| first generation h1 blocker. Mechanism: reversible inhibition of histamine receptors. uses: allergy, motion sickness, sleep aid. tx: sedation, antimuscarinic (hence the sleepy), motion sickness |
|
|
Term
|
Definition
| first generation h1 blocker. Mechanism: reversible inhibition of histamine receptors. uses: allergy, motion sickness, sleep aid. tx: sedation, antimuscarinic (hence the sleepy), motion sickness |
|
|
Term
|
Definition
| first generation h1 blocker. Mechanism: reversible inhibition of histamine receptors. uses: allergy, motion sickness, sleep aid. tx: sedation, antimuscarinic (hence the sleepy), motion sickness |
|
|
Term
|
Definition
| first generation h1 blocker. Mechanism: reversible inhibition of histamine receptors. uses: allergy, motion sickness, sleep aid. tx: sedation, antimuscarinic (hence the sleepy), motion sickness |
|
|
Term
|
Definition
| 2n generation histamine blockers, has less entry into CNS so is far less edating. used for allergy (claritan) |
|
|
Term
|
Definition
| 2n generation histamine blockers, has less entry into CNS so is far less edating. used for allergy (claritan) |
|
|
Term
|
Definition
| 2n generation histamine blockers, has less entry into CNS so is far less edating. used for allergy (claritan) |
|
|
Term
|
Definition
| 2n generation histamine blockers, has less entry into CNS so is far less edating. used for allergy (claritan) |
|
|
Term
|
Definition
| non selective Beta agonist acts on AC to increase cAMP. causes Tachycardia (b1) and broncho dialation (b2) |
|
|
Term
|
Definition
| B2 agonist relaxes bronchial smooth muscle. adverse effects are tremor and arrhythmia. |
|
|
Term
|
Definition
| B2 agonist relaxes bronchial smooth muscle. adverse effects are tremor and arrhythmia. |
|
|
Term
|
Definition
| likely causes bronchodilation by inhibiting phosophodiesterase thereby reducing cAMP hydrolysis (therby increaseing cAMP's ability to dilate. usage is limited by narrow theraputic index. metabolized by P-450 |
|
|
Term
|
Definition
| competitive block of muscarinic receptors preventing, bronchoconstriction. also used for copd. |
|
|
Term
|
Definition
| prevents released of mediators from mast cells, effective only for prophylaxis of asthma not effective during acute a attack. toxcity is rare. |
|
|
Term
|
Definition
| inhibits the synth of all cytokines. inactivates nf-kb, the transription that induces the production of TNF-a and other inflammatory agents. 1st line for chronic asthma. obviously a host of catabolic effects. |
|
|
Term
|
Definition
| 5-lipoxgenase pathway inhibitor, blocks conversion of arachadionic acid to leiotrienes |
|
|
Term
|
Definition
| blocks leukotrienes receptors, especially good for asprin induced asthma |
|
|
Term
|
Definition
| blocks leukotrienes receptors, especially good for asprin induced asthma |
|
|
Term
|
Definition
| exportant: reomves excess sputum, but large doses necessary. does not supress cough reflex. |
|
|
Term
|
Definition
| mucolytic= can loosen mucous plugs in CF patients. cleaves disulfide bonds within mucous glycoproteins. also used as an antidote for acetaminophen overdose. |
|
|