| Term 
 | Definition 
 
        | A2 is an enzyme that is a catalyst for vasocontriction. This drug resrict the body's ability to convert A1 into A2.
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        | Term 
 | Definition 
 
        | Blocks smooth muscle receptor sites in vasculature, reducing the catalyst to vasoconstrict. Increase in urine due to renal dilation.
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        | Term 
 | Definition 
 
        | Blocks neurotransmitters (norepinephrine and epinephrine) from binding. Blocking adrenelin (epinephrine) allows heart to relax.
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        | Term 
 
        | Angeotensin receptor blocker |  | Definition 
 
        | Blocks the action of A2 (the catalyst for vasoconstriction). Prescribed to patients who cannot toleate ACE inhibitors. Similar mechanics.
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        | Term 
 | Definition 
 
        | Blocks the entry of calcium into the heart and artery cells. Reduces electical conduction in heart which reduces workload/contractility.
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        | Term 
 | Definition 
 
        | Class of drugs used to suppress abnormal rhythms of the heart such as; A-Fib, A-Flutter, V-Fib, and V-Tach. 4 basic categories with affects on AV / SA nodes, Calcium, Potassium and Sodium levels |  | 
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        | Term 
 | Definition 
 
        | Water follows salt through the body. These drugs control sodium by either restricting the body's ability to reabsorb it or forcing excretion of sodium. Water follows salt out with urine.
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        | Term 
 | Definition 
 
        | Relaxes muscles surrounding the airway by activating Beta-2 receptors. Dialates the lower airway.
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        | Term 
 | Definition 
 
        | This class of drugs Reduces the amount of acid the stomach produces by blocking a important producer of acid "Histamine 2" (a common chemical in the body that signals the stomach to make acid) |  | 
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        | Term 
 | Definition 
 
        | Inhibits the final stage of gastric acid secretion. Affects specific enzymes
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        | Term 
 
        | Tryciclic Antidepressants |  | Definition 
 
        | act primarily as an SNRI by blocking both serotonin and norepinephrine transporters for reuptake, which results in an elevation in concentrations of these neurotransmitters, enhancing neurotransmission. |  | 
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        | Term 
 | Definition 
 
        | Act similar to the older TCAs (but without cardiovascular side effects), by inhibiting the reuptake of the neurotransmitters serotonin and norepinephrine, increasing neurotransmission. Have also been found to increase dopamine neurotransmission as well. |  | 
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        | Term 
 | Definition 
 
        | Inhibits or blocks the reuptake of the neurotransmitter serotonin, increasing neurotransmission and the level of serotonin available to bind to receptors. |  | 
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        | Term 
 | Definition 
 
        | Causes the body to absorb and store blood sugar as glycogen. |  | 
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        | Term 
 | Definition 
 
        | Many types and mechanisms. Most popular are designed to reduce hepatic glucose secretion and increase absorption of glucose. Normally
 taken in conjunction with insulin.
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        | Term 
 | Definition 
 
        | Almost all are over the counter medications. They act by blocking enzymes that promote pain and inflamation.
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        | Term 
 | Definition 
 
        | These drugs decrease the clotting ability of blood and help prevent harmful clots from forming in the blood vessels. These are sometimes called blood thinners, although they do not actually thin the blood. |  | 
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        | Term 
 | Definition 
 
        | Decreases platelet aggregation and inhibits the formation of clots, increasing arterial circulation when anticoagulants have little effect. |  | 
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        | Term 
 | Definition 
 
        | Lower cholesterol levels by inhibiting a cholesterol production enzyme in the liver. Also prescribed as a secondary prevention for those at risk for cardiovascular disease. |  | 
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