Term
| What is an inflammation that extends beyond the gingiva and apical migration of junctional epithelium? |
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Definition
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Term
| Would a pocket be a primary or secondary etiology factor to periodontal disease? |
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Definition
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Term
| What is a community of bacteria on an aqueous surface within a glycocalyx matrix allows increased survival/symbiosis versus bacteria in the planktonic state. |
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Definition
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Term
| What is the most prevalent type of periodontitis in adults but can occur in children and adolescents (represents bone loss mostly as a defense to bacteria)? |
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Definition
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Term
| What percentage of the US population is estimated to have periodontitis? |
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Definition
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Term
True or false:
In chronic periodontitis, amount of destruction is consistent with the presence of local factors |
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Definition
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Term
| Where will you most likely see the most plaque? |
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Definition
mandibular incisors (sublingual glands)
1st maxillary molar (parotid gland) |
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Term
What is NOT a secondary etiology of periodontitis?
Calculus
Overhanging restorations
Perio-endo lesions
Bacterial plaque
Open contacts with food impaction
Occlusal trauma
Smoking
Systemic disease |
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Definition
| bacterial plaque (primary etiology) |
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Term
| What is when a crown is prepared too deep into the where the attachment should be causing chronic inflammation (mistake of restorative dentistry)? |
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Definition
| violation of biologic width |
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Term
| In violation of biologic width, what 2 zones do we violate? |
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Definition
Zone of epithelium attached to tooth
Zone of connective tissue attached to tooth |
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Term
| Patients who smoke are ____ times more likely to have periodontal disease than nonsmokers |
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Definition
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Term
__________ is the strongest modifiable risk factor for periodontal disease.
What is the dose-dependent relationship? |
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Definition
cigarette smoking
# years
#packs/day |
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Term
| What is the severity of periodontitis based on? |
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Definition
| Based on the amount of Clinical Attachment Loss (CAL) |
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Term
| What are the measurement of clinical attachment loss for slight, moderate, and severe? |
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Definition
Slight 1-2 mm CAL
Moderate: 3-4 mm CAL
Severe: > 5 mm CAL |
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Term
| **What teeth are most severely affected by chronic periodontitis? |
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Definition
Maxillary molar >>> Mandibular canines
Interproximal sites > labial surfaces |
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Term
| What two things set aggressive and chronic periodontitis apart? |
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Definition
In aggressive,
microbial challenge can be far more virulent
host response to plaque will cause more inflammation and bone loss (low tolerance to plaque) |
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Term
| What all initiates the host-mediated destruction? |
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Definition
Cytokines and inflammatory mediators
IL-1, IL-6,IL-8, TNF-α, PGE2 |
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Term
| What are the 4 phases of host response to periodontitis? |
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Definition
1) acute bacterial challenge phase (PMNs)
2) acute inflammatory response (macrophages, inflammatory mediators)
3) immune response phase
4) regulation and resolution phase (Challenge less well controlled by host because of persistent bacterial aggression and contribution of host response modifiers (smoking, systemic disease, genetics) |
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Term
| What is a pocket formation around several/all primary teeth accompanied by bone loss and early exfoliation? |
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Definition
| prepubertal periodontitis |
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Term
| What is prepubertal periodontitis most often associated with? |
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Definition
immune disorders, usually neutrophil chemotaxis disorders were present
Some forms considered complication of systemic disease |
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Term
What is the type of periodontitis that affects permanent dentition in teenagers that have an absence of local factors (decay/filling), generally at the onset of puberty?
What teeth does it usually affect first? |
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Definition
juvenile periodontitis (or localized aggressive periodontitis)
1st molars and incisors |
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Term
| What type of periodontitis has a tendency of cases to aggregate in families? |
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Definition
| localized aggressive periodontitis |
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Term
True or False:
Aggregatibacter actinomycetemcomitans (A.a), a highly leukotoxic strain, can cause localized aggressive periodontitis alone. |
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Definition
False
some populations of patients with LAP do not harbor A. actinomycetemcomitans and in still others P. gingivalis may be etiologically more important.
It should be remembered, however, that all dental plaque- induced periodontal diseases are mixed infections and none of them is caused by a single pathogen |
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Term
| What virulence factor of A.A. forms pores in neutrophil granulocytes, monocytes, and some lymphocytes, which consequently die because of osmotic pressure? |
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Definition
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Term
| What virulence factor of A.A. inhibit neutrophil chemotaxis? |
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Definition
| Chemotactic inhibition factors |
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Term
| What virulence factor of A.A. inhibit growth of beneficial species? |
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Definition
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Term
What virulence fact of A.A. cleave IgG?
Destroys connective tissue? |
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Definition
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Term
| What population will you most likely see aggressive periodontitis? |
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Definition
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Term
| Is socioeconomic status a risk indicator or risk factor of aggressive periodontal disease? |
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Definition
| risk indicator ( it can be overcome ) |
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