Term
| The two NT's that are involved in most connections in brain (besides pain) |
|
Definition
| glutamate (exhitatory effects) and GABA (inhibitory effects). There are probably no neurons that do not recieve some exhiteatory or inhibitory input from these NT's |
|
|
Term
| What do the other NT's do besides glutamate and GABA? |
|
Definition
| have modulatory effects rather than a information transmitting effect. (i.e., activate or inhibit entire circuts of neurons involved in brain functions) |
|
|
Term
|
Definition
| acetylcholine, monoamines (dopamine, norepinephrine, serotonin), amino acids (glutamate, GABA, glycine) peptides, lipids, nuclesides, and soluble gases |
|
|
Term
|
Definition
| primary NT sercreted by efferent axons of the CNS. All muscle movement accomplished through ACh. Deactivated by acetylcholinesterase |
|
|
Term
| where is acetylcholine found in brain? |
|
Definition
| Ach is found outside brain but also inside. inside brain, found in dorsolateral pons to faciliate REM sleep, in basal forebrain to faciliate perceptual learning, and medial septum to help hippocampus with memory |
|
|
Term
|
Definition
| an acetylcholine antagonist, prevents the release of Ach by terminal buttons. This toxin is produced by a bacteria that can grow in improperly canned food |
|
|
Term
|
Definition
| a poison produced by the black widown spider that triggers the release of Ach. |
|
|
Term
|
Definition
| an ionotropic Acetylcholine receptor that is stimulated by nicotine and blocked by curare (a drug) |
|
|
Term
|
Definition
| a metobolic acetlycholine receptor that is stumilated by muscarine (a drug found in a poisonous mushroom) and blocked by atrophine (a drug) |
|
|
