Term
| what system that ang II works on makes a fast vaspopressur response? |
|
Definition
| altered peripheral resistance |
|
|
Term
|
Definition
|
|
Term
ACEI MAO does what directly?
what indirectly? |
|
Definition
increase bradykini
decease ANg I
increase ANG 1-7 |
|
|
Term
ACEI also cause an increase in _ enzyme
but a decrease in? |
|
Definition
|
|
Term
| sensitivity to any RAAS med is increased by? |
|
Definition
RAAs activity
heart failure
low Na diet |
|
|
Term
|
Definition
|
|
Term
what med affects large ateries?
how? |
|
Definition
ACEI
-increases comopliance/ decreases systolic |
|
|
Term
| what is a slight ADE of ACEI on GFR -2 |
|
Definition
lowers it
-increase serum creatitine |
|
|
Term
|
Definition
hyperkalemia
cough
angioedema
hypotension
acute renal failure in predipsoed |
|
|
Term
|
Definition
HTN
left ventricluar hypertrophy
CAD
MI
Chronic renal failure |
|
|
Term
| AT1 recptor blockers result in -3 |
|
Definition
dilation and reduced PR
reduced aldosterone
reduced sympathetic acitivty |
|
|
Term
| which med causes an increase in ANg II |
|
Definition
|
|
Term
|
Definition
HTN
heart failure
stroke prophylaxis
diabetic nephropathy |
|
|
Term
| what med inhibits the rate limitng step of RAS |
|
Definition
|
|
Term
|
Definition
headache, fatigue
diarrhea
epistais-nosebleed
hyperkal
hyperuriciema |
|
|
Term
| which med causes epsitasis? nosebleed |
|
Definition
|
|
