Term
|
Definition
| Mechanism-natural or semisynthetic agonists of the m-opoid receptor that result in inhibition of neurotransmission. |
|
|
Term
| m-opoid receptor agonist drugs |
|
Definition
Morphine
Codeine
Oxycodone
Hydrocodone |
|
|
Term
|
Definition
| Mechanism-synthetic agonists of the m-opoid receptor that results in inhibition of neurotransmission |
|
|
Term
| Synthetic m Agonists drugs |
|
Definition
Methadone
Fentanyl
Remifentanil
Meperidine
Propoxyphene |
|
|
Term
| Partial and mixed m agonists |
|
Definition
| Mechanism-partial m-receptor agonists (butorphanol and buprenorphine) and a R-agonist with partial m-antagonist activity (nalbuphine) |
|
|
Term
| Partial and mixed m agonists drugs |
|
Definition
|
|
Term
| Opoid receptor antagonists |
|
Definition
| Mechanism-antagonist of m-opoid receptor, thereby blocking endogenous or exogenous opoid effects |
|
|
Term
| Opoid receptor antagonists drugs |
|
Definition
|
|
Term
| Tricyclic antidepressants |
|
Definition
| Mechanism-promote serotonergic and noradrenergic neurotransmission by inhibiting nt reuptake |
|
|
Term
| Tricyclic antidepressants drugs |
|
Definition
Amitriptyline
Nortriptyline
Venlafaxine |
|
|
Term
| Anticonvulsants and antiarrhythmias |
|
Definition
| Mechanism-inhibit action potential initiation or conduction |
|
|
Term
| Anticonvulsants and antiarrhythmias drugs |
|
Definition
|
|
Term
| NMDA receptor antagonists |
|
Definition
| Mechanism-block NMDA receptor dependent post-synaptic depolarization |
|
|
Term
| NMDA receptor antagonists drugs |
|
Definition
|
|
