| Term 
 | Definition 
 
        | Classification: Parasympatholytic, Anticholinergic, Vagolytic Mechanism: Via direct vagolytic action enhances SA node Automaticity,  AV node Conduction used for: hemodynamically significant bradyarrhythmias and asystole to speed up heart Dose: .5mg alive,  1 mg dead  up to 3mg |  | 
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        | Term 
 | Definition 
 
        | Classification:  Vasopressor, vasoconstrictor   Mechanism: improves coronary and cerebral perfusion, Peripheral vasoconstrictor, produces favorable redistribution of blood flow during cardiac arrest   Dose: 1 mg every 3-5 min during CPR |  | 
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        | Term 
 | Definition 
 
        | Classification:  class 1B antiarrhythmic/ Ventricular antiarhythmic Mechanism:  Decreases automaticity thereby supressing ventricular arrhythmias,  elevates the V-fib threshold,  mild sodium blockage, reduces the slope of phase four diastolic repolarization Dose:  initial bolus 1-1.5 mg/kg drip 2nd bolus of .5 mg/kg after ten minutes 3rd .5 mg/kg max dose of 3mg start drip of 2-4 mg/min   |  | 
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        | Term 
 | Definition 
 
        | Classification:  Atrial  and Ventricular antiarrhythmic Mechanisms:  Effects Sodium, potassium and Calcium channels, alpha and beta adrenergic blockage, coronary and peripheral vasodilation, lengthens refractory period in cardiac tissue Dose:  300 mg IV Bolus if dying otherwise 150mg over 10 min(may be repeated as needed) max dose 2gm/day |  | 
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        | Term 
 | Definition 
 
        | Type: Ventricular Antiarrhythmic Mechanism:  supresses ventricular ectopy, slows intraventricular conduction extends phase four of action potential Dose: 20-30 mg/min up to 17mg/kg maintenance rate is 1-4mg/min |  | 
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        | Term 
 | Definition 
 
        | Type: Ventricular antiarrhythmic Mechanisms:  reduction in post infarction ventricular arrhythmias, precipitate refractory v-fib, hypomagnesemia can lead to cardiac arrhythmias and sudden cardiac death Dose:  V-fib or V-tach-1-2gm in 100ml NS over 1-2 min Post MI:  loading dose 1-2mg in 100 ml over 5-60 min |  | 
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        | Term 
 | Definition 
 
        | Type: Supraventricular antiarrhythmic Mechanism: bring heart back to normal rhythm,not useful for all types of irregular heartbeats, slows AV node conduction, terminated PSVT,  May be used to test the heart for coronary artery disease, has a half-life of 5 seconds, not a fix for A-fib or A-flutter but a great diagnostic tool Dose: 6mg over 1-3 sec followed by 20ml flush 12mg over 1-3 sec 12mg over 1-3 sec |  | 
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        | Term 
 | Definition 
 
        | Type: Supraventricular anti-arrhythmic, Calcium channel blocker Mechanisms:  Calcium channel Blocker,  Relaxs blood vessels,  Vasodilatoreffects due to blockade of Ca channels and alpha receptors, decreases inotropy Dose: initial 2.5 - 5.0 mg IV over 2 min repeat dose 5 -10 mg 15-30 min until max of 20mg |  | 
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        | Term 
 | Definition 
 
        | Type: Supraventricular Antiarrhythmic, Ca channel blocker Mechanism: fewer hemodynamic effects than verapamil, slows conduction and prolongs refractoriness in AV node Dose: .25 mg/kg over 2 min followed by maintenance infusion of 5-15mg titrated to desired heart rate |  | 
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        | Term 
 | Definition 
 
        | Type: Ca++ channel blocker Mechanism: Dilates main coronary arteries and arterioles in both normal and ischemic areas of the heart, inhibits spasms of the coronary arteries Dose: 10-20mg TID to control angina Max 180mg/day |  | 
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