Term
| 1. The 3 layers of arteries are the _____, _____ and the _____. |
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Definition
| TUNICA INTIMA, TUNICA MEDIA, TUNICA ADVENTITIA |
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Term
| 2. The _____ layer of arteries is where lesions and fatty deposits mostly occur. |
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Definition
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Term
| 3. 3 components of the tunica intima layer of arteries. |
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Definition
| 1) ENDOTHELIUM, 2) SUBENDOTHELIUM, 3) ELASTIC LAYER |
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Term
| 4. The _____ layer of blood vessels is partially where lesions occur. |
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Definition
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Term
| 5. 2 components of the tunica media layer of arteries. |
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Definition
| 1) SMOOTH MUSCLE, 2) ELASTIC FIBERS |
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Term
| 6. What makes up the tunica adventitia layer of arteries? |
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Definition
| LOOSE CONNECTIVE TISSUE WITH NOURSIHING VESSELS (VASA VASORUM) |
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Term
| 7. 6 components of the recurrent injury hypothesis of atherosclerosis. |
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Definition
| 1) RECURRENT INJURY, 2) ENDOTHELIAL DISRUPTION, 3) MONOCYTE ADHERENCE AND ACTIVATION, 4) SMOOTH MUSCLE CELL PROLIFERATION, 5) ACCUMULATION OF LIPIDS, 6) ORGANIZATION OF MATURE PLAQUE |
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Term
| 8. _____ can occur beneath the endothelium of arteries, and can easily disrupt and cause almost immediate clotting. |
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Definition
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Term
| 9. 2 alternatives to statin drugs are _____ and _____ which can help with cardio health. |
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Definition
| CHINESE RED RICE EXTRACT, POLYCOSANOLS |
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Term
| 10. Because lipids are insoluble in plasma, they are encapsulated by certain fat-carrying proteins called _____. |
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Definition
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Term
| 11. 5 different classes of lipoproteins. |
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Definition
| 1) CHYLOMICRONS, 2) VLDL’S, 3) IDL’S. 4) LDL’S, 5) HDL’S |
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Term
| 12. _____ are lipoproteins synthesized by the small intestine and liver. They are involved in the transport of exogenous triglycerides and cholesterol that has been absorbed from the GI tract. |
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Definition
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Term
| 13. _____ are lipoproteins synthesized by the liver. They are the primarily endogenous pathway for transform of triglycerides that are already in the body. |
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Definition
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Term
| 14. VLDL’s carry triglycerides to tissue capillaries for entry into _____ and _____ cells. |
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Definition
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Term
| 15. _____ constitute the lipoprotein remnants that remain when the triglycerides are removed from the VLDL. |
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Definition
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Term
| 16. IDL’s are either taken up and broken down by the _____, or they are converted to _____. |
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Definition
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Term
| 17. _____ are the major carriers of cholesterol, so its measurements provide good estimates of blood cholesterol. |
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Definition
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Term
| 18. About 2/3 of the circulating LDL’s are removed by the _____ by means of the LDL receptors. The remainder is taken up and broken down in peripheral tissues by degradative process which includes _____. |
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Definition
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Term
| 19. When macrophages uptake LDL within the arterial wall, it can result in these 3 things. |
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Definition
| 1) ACCUMULATION OF ESTER, 2) FORMATION OF FOAM CELLS, 3) DEVELOPMENT OF ATHEROSCLEROSIS |
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Term
| 20. _____ serve as the carriers that remove cholesterol from the peripheral tissue, and transport it back to the liver for catabolism and excretion. They also inhibit cellular uptake of LDL’s. |
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Definition
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Term
| 21. High levels of _____ contribute to the development of artherosclerosis, whereas high levels of _____ appear to provide some protection against the disorder. |
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Definition
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Term
| 22. 3 groups of people who have elevated levels of HDL’s. |
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Definition
| 1) PEOPLE WHO EXERCISE REGULARLY, 2) WOMEN WHO TAKE ESTROGENS, 3) PEOPLE WHO CONSUME MODERATE AMOUNTS OF ALCOHOL |
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Term
| 23. _____ and _____ are associated with high levels of LDL’s and low levels of HDL’s. |
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Definition
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Term
| 24. LDL’s are the major carriers of _____, and form _____. |
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Definition
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Term
| 25. VLDL’s are the major carriers of _____. |
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Definition
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Term
| 26. HDL’s are the good guys in that they prevent _____ formation. |
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Definition
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Term
| 28. 5 major risk factors of cardiovascular disease that can be changed. |
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Definition
| 1) CIGARETTE SMOKING, 2) HIGH BP, 3) BLOOD CHOLESTEROL LEVELS, 4) DIABETES, 5) STRESS |
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Term
| 29. 4 major risk factors of cardiovascular disease that cannot be changed. |
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Definition
| 1) HEREDITY, 2) SEX, 3) RACE, 4) AGE |
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Term
| 30. _____ is the core/root of angina. |
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Definition
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Term
| 31. This type of ischemia does not have symptoms or angina. |
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Definition
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Term
| 32. If a person presents with ischemic hypoxia, their ECG will show _____ and might have a _____. |
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Definition
| ST SEGMENT DEPRESSION, T WAVE INVERSION |
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Term
| 34. In this type of hypoxia, there is a lack of O2 to cells due to a lack of oxygen loading in the blood. |
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Definition
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Term
| 35. In this type of hypoxia there is a lack of blood flow due to restricted O2 causing cellular swelling. |
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Definition
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Term
| 36. If a person has ischemic hypoxia, their cells will lose the ability to do these 2 things. |
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Definition
| 1) LOSE THE BAILITY TO SYNTHESIZE PROTEINS, 2) LOSE CELL MEMBRANE PERMEABILITY |
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Term
| 37. This type of hypoxia is caused by a lack of O2 to cells due to decreased RBCs and/or hemoglobin. |
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Definition
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Term
| 38. This type of hypoxia is caused by a toxin that caused oxygen deprivation in the tissues. |
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Definition
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Term
| 39. _____ is the number one cause of cell injury. |
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Definition
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Term
| 40. When cell injury is sustained due to hypoxia, _____ keeps the platelets from getting sticky, and sometimes it removes the clot and restores the flow of blood in the lumen. |
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Definition
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Term
| 41. When is cell intervention too late/ what is the point of no return? |
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Definition
| WHEN MITOCHONDRIA FILL WITH CALCIUM, WHICH DESTROYS ATP MAKERS |
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Term
| 42. O2 is necessary to manufacture _____ in cardiac cells. |
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Definition
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Term
| 43. When cardiac cells are in O2 deprivation, it goes anaerobic for a while and causes a build up of _____. |
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Definition
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Term
| 44. Eventually the lack of ATP will cause _____ failure. |
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Definition
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Term
| 45. When there is NA/K pump failure, sodium and water accumulate inside the cell causing _____. |
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Definition
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Term
| 46. _____ is an early sign of cell injury. |
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Definition
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Term
| 47. Cellular swelling is _____. |
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Definition
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Term
| 48. When cellular swelling occurs, organelles become distorted, disrupted, and porous affecting the cell membrane in these 2 ways. |
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Definition
| 1) RIBOSOMES CAN’T MANUFACTURE PROTEINS BECAUSE OF DISTORTION, 2) POROSITY OF MEMBRANE WILL ALLOW CALCIUM TO ACCUMULATE IN THE CELL WHICH IS A HALLMARK OF CELL INJURY, 3) MITOCHONDRIA BURST |
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Term
| 49. 4 signs and symptoms of hypoxia. |
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Definition
| 1) DIAPHORESIS, 2) ANXIETY, 3) GREAT PAIN, 4) NAUSEA |
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Term
| 50. If there is cellular injury occurring an ECG will show a _____ and _____. |
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Definition
| ST ELEVATION AND WIDENED Q WAVE |
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Term
| 51. If cellular injury occurs, blood work will show up _____ hours after cell injury. |
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Definition
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Term
| 52. If cellular injury occurs the blood work will show an elevation of these 5 cardiac enzymes. |
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Definition
| 1) CK, 2) CPK, 3) CPK-MB, 4) SGOT/AST, 5) LDH |
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Term
| 53. _____ begins rising 3-4 hours after cellular injury. |
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Definition
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Term
| 54. Is found in the actin and myosin of muscle cells, and begins rising 4-6 hours after cellular injury. |
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Definition
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Term
| 55. _____ is responsible for O2 storage in muscle cells, and begins rising 1-2 hours after injury. |
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Definition
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Term
| 56. _____ begins rising 4-6 hours after cellular injury. |
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Definition
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Term
| 57. If there is cellular injury WBC count will be _____ within 12-24 hours. _____ is usually due to systemic infection with is part of the inflammatory response. |
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Definition
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Term
| 58. 4 manifestation categories of a myocardial infarction. |
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Definition
| 1) PAIN AND AUTONOMIC RESPONSES ASSOCIATED WITH ISCHEMIC EVENTS, 2) WEAKNESS AND SIGNS RELATED TO IMPAIRED MYOCARDIAL FUNCTION, 3) DYSRHYTHMIA, 4) ECG CHANGES |
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Term
| 59. 3 ECG changes that will occur in a patient with a MI. |
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Definition
| 1) ST ELEVATION, 2) T WAVE INVERSION, 3) WIDE Q WAVE |
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Term
| 60. The ECG changes that occur in a patient with an MI are associated with _____ and _____ of myocardial cells. |
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Definition
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Term
| 61. 7 complications of an MI. |
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Definition
| 1) SUDDEN DEATH, 2) HEART FAILURE, 3) PERICARDITIS, 4) THROMBOEMBOLI, 5) RUPTURE OF MYOCARDIUM, 6) ANEURYSM, 7) ARRHYTHMIAS |
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Term
| 62. Pain from myocardial infarctions present as _____ and _____ pain. |
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Definition
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Term
| 63. The vagal stimulation from myocardial infarction will cause _____ and _____. |
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Definition
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Term
| 64. The sympathetic stimulation from myocardial infarction will cause _____, _____ and a _____. |
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Definition
| TACHYCARDIA, ANXIETY, AND AN INCREASE IN BP |
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Term
| 65. Following a myocardial infarction, no tissue changes are noted in the first _____ hours. |
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Definition
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Term
| 66. After _____ hours, the tissue turns pale to grey-brown, with slight pallor of the area. |
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Definition
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Term
| 67. After _____ days following a myocardial infraction, necrosis of the area is apparent In the center and hyperemic around the edges. |
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Definition
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Term
| 68. After _____ days following a myocardial infarction, the area becomes soft and fatty changes in the center are well developed. Hemorrhagic areas are present in the infarcted areas. |
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Definition
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Term
| 69. After _____ days following a myocardial infarction, fibrotic scar tissue replacement occurs and revascularization commences. |
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Definition
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Term
| 70. After _____ weeks following a myocardial infarction, scar tissue replacement of necrotic tissues ensues. |
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Definition
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Term
| 71. _____ is the number 1 killer of people in the US. It is a disease that manifests within the lumen of the coronary arteries, or beneath the endothelium. |
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Definition
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Term
| 72. _____ is the most common artery affected by coronary artery disease. |
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Definition
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Term
| 73. _____ is the major symptom of coronary artery disease. |
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Definition
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Term
| 74. 3 types of angina that present in coronary artery disease. |
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Definition
| 1) CLASSIC ANGINA, 2) UNSTABLE ANGINA, 3) VARIABLE ANGINA |
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Term
| 75. _____ is angina that is caused by an increased workload on the heart during physical stress. It often goes away after rest. |
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Definition
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Term
| 76. _____ is angina that is 1 step closer to cell injury and infarction. There are periods of some blocking where it cuts off the blood flow beyond the lesion. During this period of time there can be partial or total obstruction. |
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Definition
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Term
| 77. Unstable angina is not associated with exercise, rather it comes during _____. |
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Definition
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Term
| 78. _____ is a vasodilator used with unstable angina that is a vasodilator used to bring relief of symptoms by improving oxygen supply. |
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Definition
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Term
| 79. _____ is vasospastic angina not usually associated with atherosclerosis. Instead it is associated with spastic vessels. |
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Definition
| VARIABLE ANGINA (PRINZMETAL’S ANGINA) |
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Term
| 80. _____ will be the final outcome of variable angina in the _____ arteries. |
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Definition
| HYPOXIA; CORONARY ARTERIES |
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Term
| 81. _____ is fibrous banding that encases the heart like a cast. The heart can’t fill or pump very well, causing a life threatening situation. |
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Definition
| CONSTRICTIVE PERICARDITIS |
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Term
| 82. 6 impaired cardiac functions that can cause heart failure. |
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Definition
| 1) CARDIOMYOPATHY, 2) MI, 3) CORONARY INSUFFICIENCY, 4) CONGENITAL DEFECTS, 5) CONSTRICTIVE PERICARDITIS, 6) VULVAR HEART DISEASE |
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Term
| 83. 3 excess work demands of the heart that can eventually lead to heart failure. |
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Definition
| 1) INCREASED PRESSURE CAUSING STRICTURING OF THE AORTA, 2) INCREASED VOLUME WORK, 3) INCREASED PERFUSION WORK |
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Term
| 84. _____ causes congestive blood back up into the lungs eventually leading to pulmonary edema. |
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Definition
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Term
| 85. If left sided heart failure is present in a patient, a doctor will hear _____ or _____, and the patient might also present with _____ because of collapsed alveoli. |
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Definition
| RALES, CRACKES; ABSENT BREATH SOUNDS |
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Term
| 86. If a patient presents with left sided heart failure, the backup will go all the way back and stress the _____. |
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Definition
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Term
| 87. The number one cause of right sided heart failure is _____. |
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Definition
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Term
| 88. If left sided heart failure causes right sided heart failure, the venous blood will back up to the jugular veins and one will see _____. |
|
Definition
| JUGULAR VENOUS DISTENTION |
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Term
| 89. 7 signs and symptoms of a patient with left sided heart failure. |
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Definition
| 1) EXERTIONAL DYSPNEA, 2) ORTHOPNEA, 3) PAROXYSMAL NOCTURNAL DYSPNEA, 4) PRODUCTIVE COUGH, 5) BLOOD-TINGED SPUTUM, 6) CYANOSIS, 7) ELEVATION IN PULMONARY CAPILLARY WEDGE PRESSURE |
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Term
| 90. To test elevation in pulmonary capillary wedge pressure during left sided heart failure use a _____, which is threaded through the right ventricle, pulmonary trunk, and it measures the left ventricular pressure. |
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Definition
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Term
| 91. 3 treatments used for left sided heart failure that can also help right sided heart failure. |
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Definition
| 1) DIGOXIN, 2)ACE INHIBITOR, 3) BIODIN FOR AFRICAN AMERICANS |
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Term
| 92. 9 symptoms of right sided heart failure. |
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Definition
| 1) HEPATOMEGALY, 2) SPLENOMEGAY, 3) PORTAL HYPERTENSION, 4) FATIGUE, 5) DEPEDENT EDEMA, 6) JUGULAR VEIN DISTENSION, 7) ANOREXIA, 8) CYANOSIS, 9) PERIPHERAL VENOUS PRESSURE |
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Term
| 93. 3 bodily compensatory mechanisms when tissues are not getting enough O2. |
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Definition
| 1) SYMPATHETIC NERVOUS SYSTEM, 2) RENIN-ANGIOTENSIN-ALDOSTERONE, 3) ANAEROBIC METABOLISM |
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Term
| 94. 4 responses of the sympathetic nervous system in response to tissues not getting enough O2. |
|
Definition
| 1) INCREASES HEART RATE, 2) INCREASED MYOCARDIAL CONTRACTILITY, 3) INCREASED WORK DEMAND ON THE HEART, 4) VASOCONSTRICTION LEADING TO INCREASED PVR |
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Term
| 95. 4 ways the renin-angiotensin-aldosterone system compensates in response to tissues not getting enough O2. |
|
Definition
| 1) JGA CELLS DETECT A DECREASE IN BLOOD PRESSURE AND RELEASE RENIN, 2) RENIN IS CONVERTED TO ANGIOTENSINOGEN IN THE LIVER WHICH CONVERTS TO AG2, 3) THIS CAUSES THE BODY TO RETAIN MORE SODIUM AND WATER INCREASING BLOOD VOLUME, 4) THE INCREASED VOLUME INCREASES THE WORK LOAD ON THE HEART INCREASING PVR |
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Term
| 96. 3 steps in how anaerobic metabolism compensated in response to tissues not getting enough O2. |
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Definition
| 1) O2 NOT GETTING TO CELLS DECREASES ATP PRODUCTION, 2) THIS CAUSES A BUILD UP OF LACTIC ACID, 3) ACIDOSIS IMPAIRS CONTRACTILITY OF THE HEART |
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Term
| 97. 9 steps in cell injury |
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Definition
| 1) DECREASED O2 TO CELLS, 2) DECREASED ATP SYNTHESIS, 3) FAILURE OF THE NA/K PUMP, 4) SODIUM ACCUMULATES INSIDE THE CELL CAUSING WATER TO FLOW IN, 5) CELLULAR SWELLING INSUES, 6) THIS CAUSES CELLULAR DISTORTION DAMAGING ORGANELLES, 7) PROTEIN SYNTHESIS DECLINES, 8) CELL MEMBRANES BECOME PERMEABLE, 9) CALCIUM ENTERS THE CELL DESTROYING MITOCHONDRIA |
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Term
| 98. 8 noninvasive assessments of cardiovascular function. |
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Definition
| 1) SENSORIUM, 2) MUCOUS MEMBRANE COLOR, 3) PULSE RATE, 4) AUSCULTATION OF HEART SOUNDS, 5) CARDIOGRAPHY, 6) DOPPLER STUDIES, 7) STRESS TESTING, 8) CHEST X-RAYS |
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Term
| 99. 3 invasive assessments of cardiac function. |
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Definition
| 1) NUCLEAR IMAGING WITH RADIOLABELED PHARMACEUTICALS, 2) CARDIAC CATHERIZATION, 3) CORONARY ANGIOGRAPHY |
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Term
| 100. 4 causes of impaired cardiac function. |
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Definition
| 1) MYOCARDIAL DISEASE, 2) VALVAR HEART DISEASE, 3) CONGENITAL HEART DEFECTS, 4) CONSTRICTIVE PERICARDITIS |
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Term
| 101. 4 myocardial diseases that cause impaired cardiac function. |
|
Definition
| 1) CARDIOMYOPATHIES, 2) MYOCARDITIS PULMONARY HYPERTENSION, 3) CORONARY INSUFFICIENCY, 4) MI |
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Term
| 102. 2 valvar heart diseases that cause impaired cardiac function. |
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Definition
| 1) STENOTIC VALVULAR DISEASE, 2) REGURGITATION VULVAR DISEASE |
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Term
| 103. 3 causes of increased pressure work of the heart. |
|
Definition
| 1) SYSTEMIC HYPERTENSION, 2) PULMONARY HYPERTENSION, 3) COARCTATION OF THE AORTA |
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Term
| 104. 2 causes of increased volume work of the heart. |
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Definition
| 1) ARTERIOVENOUS SHUNT, 2) EXCESS IV FLUIDS |
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Term
| 105. 2 causes of increased perfusion work of the heart. |
|
Definition
| 1) THYROTOXICOSIS, 2) ANEMIA |
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Term
| 106. 5 etiologies of congestive cardiomyopathy. |
|
Definition
| 1) MYOCARDIAL DAMAGE CAUSED BY DRUGS, 2) ALCOHOLISM, 3) PERIPARTUM CARDIOMYOPATHY, 4) FOLLOWING A PREVIOUS INFECTION, 5) INEFFICIENT PUMP |
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Term
| 107. The most common manifestations of cardiomyopathy are _____ and _____. |
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Definition
|
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Term
| 108. 5 manifestations of congestive cardiomyopathy. |
|
Definition
| 1) DYSPNEA AND FATIGUE, 2) PALPATIONS ASSOCIATED WITH ARRHYTHMIAS, 3) CHEST PAIN, 4) ELEVATED BP, 5) EXTRA HEART SOUNDS AND CARDIAC MURMURS |
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Term
| 109. The prognosis of congestive cardiomyopathy is _____. |
|
Definition
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Term
| 110. 6 treatments of congestive cardiomyopathy. |
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Definition
| 1) SALT RESTRICTION, 2) DIGITALIS GLYCOSIDES AND DIURETICS, 3) ANTICOAGULANTS, 4) BED REST, 5) CORTICOSTEROIDS, 6) VASODILATORS |
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Term
| 111. _____ is a condition caused by an increased interventricular wall septal thickness causing obstruction. |
|
Definition
| HYPERTROPHIC CARDIOMYOPATHY |
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Term
| 112. If one has hypertrophic cardiomyopathy, there will be an obstructive outcome during _____. |
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Definition
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Term
| 113. 3 manifestations of hypertrophic cardiomyopathy. |
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Definition
| 1) ANGINA, 2) DIZZINESS, 3) CHF |
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Term
| 114. The 2 therapies of hypertrophic cardiomyopathy is either _____ or _____. |
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Definition
|
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Term
| 115. _____ is usually caused by an infiltrative disease of the myocardium such as amyloidosis or hemochromatosis causing a stiff non-compliant heart. |
|
Definition
| RESTRICTIVE CARDIOMYOPATHY |
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Term
| 116. The most common clinical manifestation is _____. |
|
Definition
| RIGHT SIDED CONGESTIVE HEART FAILURE |
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Term
| 117. If one has restrictive cardiomyopathy, the myocardium becomes _____ and _____. |
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Definition
|
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Term
| 118. If one has restrictive cardiomyopathy, _____ is impeded and filling pressures are raised during diastole. |
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Definition
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Term
| 119. If one has restrictive cardiomyopathy, death occurs as a result of _____ or _____. |
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Definition
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Term
| 120. _____ is a group of diseases that result from the abnormal deposition of a particular protein called amyloid in various tissues of the body. |
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Definition
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Term
| 121. When amyloidosis occurs in a single tissue of the body is it called _____. |
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Definition
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Term
| 122. When amyloidosis occurs in multiple tissues in the body it is called _____, and it can cause serious changes in virtually any organ of the body. |
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Definition
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Term
| 123. A _____ is an out-pouching/bubbling of an artery. |
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Definition
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Term
| 124. A _____ aneurysm is caused by a clot. |
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Definition
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Term
| 125. _____ is an aneurysm when there is a tear in the aortic wall through the tunica intima and perhaps the media. |
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Definition
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Term
| 126. If a dissecting aneurysm results, a patient will die within _____ hours. |
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Definition
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Term
| 127. The most common artery affected by an aneurysm is the _____. |
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Definition
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Term
| 128. Abdominal aortic aneurysms most commonly occurs _____. |
|
Definition
| AT THE BIFURCATION OF THE ABDOMINAL AORTA INTO THE COMMON ILIAC ARTERIES |
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Term
| 129. The most common artery affected by a dissecting aneurysm is the _____. |
|
Definition
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Term
| 130. 7 symptoms of a thoracic aneurysm. |
|
Definition
| 1) SUBSTERNAL, BACK AND NECK PAIN, 2) DYSPNEA AND STRIDOR, 3) TRACHEAL PRESSURE, 4) HOARSENESS OF VOICE, 5) DIFFICULTY SWALLOWING, 6) DISTENSION OF THE NECK DUE TO SVC COMPRESSION, 7) AORTIC REGURGITATION |
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Term
| 131. If one has an abdominal aneurysm, a _____ may provide the first evidence of the disorder. |
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Definition
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Term
| 132. _____ which usually exists on the wall of an aneurysm may be detected during abdominal x-ray examinations. |
|
Definition
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Term
| 133. If one has an abdominal aneurysm, pain may be present in the _____ and _____. |
|
Definition
| MID-ABDOMEN TO LOWER BACK |
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Term
| 134. If one has an abdominal aneurysm, the aneurysm may extend to and impinge the _____, _____, or _____ arteries. |
|
Definition
| RENAL, ILIAC, OR MESENTERIC |
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Term
| 135. If one has an abdominal aneurysm, stasis of blood favors _____ along the wall of the vessel and _____ may develop causing symptomatic arterial insufficiency. |
|
Definition
| THROBUS FORMATION; PERIPHERAL EMBOLI |
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Term
| 136. Could help rule out adjusting a patient with an abdominal aneurysm. |
|
Definition
|
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Term
| 137. _____ is an acute life threatening condition that involves a hemorrhage into the vessel wall with longitudinal tearing of the vessel wall to form a blood-filled channel. |
|
Definition
|
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Term
| 138. Dissecting aneurysm is caused by conditions that weaken or cause degenerative changes in the elastic and smooth muscle layer of the _____. |
|
Definition
| MEDIAL LAYER OF THE AORTA |
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Term
| 139. Dissecting aneurysms most commonly affect _____ aged _____. |
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Definition
|
|
Term
| 140. 90% of dissecting aneurysm cases are associated with a previous history of _____. |
|
Definition
|
|
Term
| 141. The most common site of a dissecting aneurysm is the _____, while the second most common site is the _____. |
|
Definition
| ASCENDING AORTA; THORACIC AORTA JUST DISTAL TO THE ORIGIN OF THE SUBCLAVIAN ARTERY |
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Term
| 142. The major symptom of a dissecting aneurysm is an abrupt appearance of excruciating pain described as _____or _____. |
|
Definition
|
|
Term
| 143. Within the first 48 hours, _____% of people die who have a untreated dissecting aneurysm. |
|
Definition
|
|
Term
| 144. _____% of untreated dissecting aneurysm patients die within 6 weeks. |
|
Definition
|
|
Term
| 145. _____ is idiopathic high blood pressure that usually goes undetected due to a lack of symptoms. |
|
Definition
| PRIMARY/ESSENTIAL HYPERTENSION |
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|
Term
| 146. Primary/essential hypertension represents _____% of hypertension cases. |
|
Definition
|
|
Term
| 147. 4 causes of secondary hypertension. |
|
Definition
| 1) CUSHINGS DISEASE, 3) PRIMARY ALDOSTERONISM, 3) PHEOCHROMOCYTOMA, 4) COARCTATION OF THE AORTA |
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Term
| 148. 9 risk factors of hypertension. |
|
Definition
| 1) FAMILY HISTORY, 2) AGE, 3) MALE GENDER, 4) BLACK RACE, 5) OBESITY, 6) HIGH SODIUM DIET, 7) GLUCOSE INTOLERANCE, 8) SMOKING, 9) HEAVY ALCOHOL CONSUMPTION |
|
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Term
| 149. What is the best way to diagnose hypertension. . |
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Definition
| TAKE BP AT THE SAME TIME OF DAY AND RECORD IT OVER A PERIOD OF DAYS |
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Term
| 150. If one has hypertension, there will be ophthalmic changes in the _____ of the eyes. |
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Definition
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Term
| 151. _____ is thong term effect of hypertension, so we should run kidney function tests when one presents with hypertension. |
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Definition
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Term
| 152. If one has hypertension, there will be a _____ in an ECG. |
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Definition
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Term
| 153. 7 steps in the pathyphysiology of hypertension. |
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Definition
| 1) HYPERTENSION DAMAGES THE WALLS OF THE SYSTEMIC VESSELS, 2) PROLONGED VASOCONSTRICTION AND HIGH PRESSURE CAUSES THE VESSELS TO THICKEN, 3) ARTERIAL SMOOTH MUSCLE UNDERGOES HYPERTROPHY, 4) TUNICA INTIMA AND MEDIA UNDERGO FIBROMUSCULAR THICKENING LEADING TO NARROWING OF THE LUMEN, 5) VESSELS BECOME PERMANENTLY NARROWED, 6) HYPERTENSIVE INJURY CAUSES INFLAMMATION TO INCREASE THE PERMEABILITY OF THE VASCULAR ENDOTHELIUM, 7) AS PERMEABILITY INCREASES MORE IONS ENTER THE VESSEL WALLS CAUSING FURTHER THICKENING |
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Term
| 154. When _____ occur, hypertension is usually far advanced. |
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Definition
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Term
| 155. 7 manifestations of hypertension. |
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Definition
| 1) UNSTEADINESS, 2) WALKING HEADACHE, 3) BLURRED VISION, 4) DEPRESSION, 5) RENAL DYSFUNCTION, 6) RETINAL CHANGES, 7) SIGNS OF VENTRICULAR FAILURE SUCH AS DYSPNEA OR EXERTION |
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Term
| 156. 4 hypertensive death related causes. |
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Definition
| 1) STROKE, 2) MI, 3) RENAL FAILURE, 4) ENCEPHALOPATHY |
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Term
| 157. _____ is a CVA when a blood clot lodges in a vessel leading to ischemic hypoxia. |
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Definition
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Term
| 158. _____ is a CVA when a clot comes from a far away source. |
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Definition
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Term
| 159. _____ is a CVA caused by bleeding. |
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Definition
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Term
| 160. Hypertension is the primary cause of _____ strokes. |
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Definition
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Term
| 161. _____ are small stroke like incidents that last 10 minutes or less. It leads to a loss in a patients senses. |
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Definition
| TIA (TRANSIENT ISCHEMIC ATTACK) |
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Term
| 162. If one has a TIA, a patient will experience these 2 symptoms. |
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Definition
| 1) BRUITS, 2) THEY WILL SEE FLOATERS |
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Term
| 163. For something to be diagnosed as a stroke, symptoms must remain for _____ hours. |
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Definition
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Term
| 164. If a patient has a thrombotic stroke, _____ can be administered to bust out the clot that’s blocking the brain, and restore blood flow. This is able to completely resolve the stroke leading too complications. |
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Definition
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Term
| 165. Strokes tame time to _____ to completion. It might not look severe at first, but the next day the patient could look much worse. |
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Definition
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Term
| 166. Once a patient has completed a stoke they should work on _____ as quick as possible. |
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Definition
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Term
| 167. _____ and _____ are 2 agents used to treat thrombotic and embolic strokes. They must not be administered to a patient with a hemorrhagic stroke because they will bleed to death. |
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Definition
| FIBRINOLYSIS AND FIBRINOLYTIC AGENTS |
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Term
| 168. 13 risk factors of cerebrovascular disease. |
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Definition
| 1) ATHEROSCLEROSIS, 2) HYPERTENSION, 3) HIGH CHOLESTEROL, 4) FAMILY HISTORY, 5) OBESITY, 6) SEDENTARY LIFESTYLE, 7) ORAL CONTRACEPTIVES, 8) SMOKING, 9) COAGULATION DISORDERS, 10) DEHYDRATION, 11) CHRONIC HYPOXIA, 12) CIGARETTE SMOKING |
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Term
| 169. 4 manifestations of a left hemispheric stroke. |
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Definition
| 1) RIGHT SIDED HEMIPLEGIA, 2) SLOW AND CAUTIOUS BEHAVIOR, 3) RIGHT VISUAL FIELD DEFECT, 4) EXPRESSIVE, RECEPTIVE OR GLOBAL DYSPHASIA |
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Term
| 170. 7 manifestations of a right hemispheric stroke. |
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Definition
| 1) LEFT SIDED HEMIPLEGIA, 2) SPATIAL-PERCEPTUAL DEFICITS, 3) IMPULSIVE BEHAVIOR, 4) DISTRACTABILITY, 5) DENIAL, 6) LEFT VISUAL FIELD DEFECTS, 7) PERSEVERATION (REPEATING A WORD OVER AND OVER) |
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