Term
| WHAT MEDICATIONS WORK WITH CLASS II? WHAT ARE THEY USED TO PREVENT? |
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Definition
| BETA BLOCKERS. USED TO PREVENT AND TREAT SUPRAVENTRICULAR AND VENTRICULAR ARRHYTHMIAS |
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Term
| WHAT DOES CLASS II INHIBIT TO DECREASE AUTOMATICITY AND CONDUCTION VELOCITY. |
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Definition
| SYMPATHETIC EFFECTS. THIS DECREASES RATE OF RISE FOR SA NODE DEPOLARIZATION, DECREASE AV CONDUCTION AND INCREASE AV REFRACTORY PERIOD... LIKE DIG. |
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Term
| WHAT DOES CLASS II HAVE LESS EFFECT ON (HINT CONDUCTION) AND MORE EFFECT ON (HINT POTENTIALS)? |
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Definition
| LESS EFFECT ON VENTRICULA CONDUCTION AND MORE EFFECT ON NODAL POTENTIALS. |
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Term
| WHAT ARE THE 3 MOST COMMONLY USED BETA BLOCKERS FOR CLASS II? |
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Definition
| METOPROLOL, ESMOLOL, AND PROPANOLOL |
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Term
| WHAT IS ANOTHER BETA BLOCKER THAT CAN ACTUALLY WORK IN ANOTHER CLASS? |
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Definition
| SOTALOL. BLOCKS K CHANNELS ALSO SO IT IS ACTUALLY A CLASS II AND CLASS III. |
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Term
| WHAT IS A NON-SELECTIVE BETA BLOCKER THAT ALSO PROLONGS ACTION POTENTIAL DURATION AND WTI BY BLOCKING POTASSIUM CHANNELS (PHASE 3 PROLONGED). |
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Definition
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Term
| SOTALOL IS USED FOR TREATMENT OF WHAT 2 ARRHYTHMIAS? |
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Definition
| VENTRICULAR AND ATRIAL ARRHYTHMIAS. |
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Term
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Definition
| BLOCKS K CHANNELS THAT ARE RESPONSIBLE FOR REPOLARIZATION DURING STAGE 3. THIS CAUSES A LONGER PLATEAU DURATION. |
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Term
| IF CLASS III INCREASES THE REFRACTORY PERIOD WHAT DOES THAT DO TO REENTRY MECHANISMS? |
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Definition
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Term
| CAN CLASS III CONTRIBUTE TO THE DEVELOPMENT OF AFTER DEPOLARIZATIONS? |
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Definition
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Term
| TYPICALLY CLASS III DOESN'T SLOW VENTRICULAR CONDUCTION VELOCITY. WHAT MEDICATION IS AN EXCEPTION? |
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Definition
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Term
| WHAT ARE 4 DRUGS THAT WORK ON CLASS 4? |
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Definition
| IBUTILIDE, DOFETILIDE, BRETYLIUM, AMIODARONE |
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Term
| WHAT TYPE OF ARRHYTHMIAS IS IBUTILIDE USED TO TERMINATE? |
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Definition
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Term
| HOW DOES IBUTILIDE WORK LIKE OTHER CLASS III DRUGS? |
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Definition
| PROLONGS REPOLARIZATION BY INHIBITING K DURING PHASE 3. |
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Term
| WHAT KIND OF METABOLISM DOES IBUTILIDE HAVE? |
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Definition
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Term
| WHAT IS THE HALF LIFE OF IBUTILIDE AND WHAT ARRHYTMIA CAN IT INDUCE? |
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Definition
| 6 HRS. CAN INDUCE TORSADES |
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Term
| WHAT IS THE MAIN DIFFERENCE WITH DOFETILIDE? |
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Definition
| SIMILAR TO IBUTILIDE EXCEPT CAN BE GIVEN PO. |
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Term
| WHAT 2 THINGS IS BRETYLIUM USEFUL FOR? |
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Definition
| USEFUL AS AN ANTIHYPERTENSIVE AND ANTIARRHYTHMIC |
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Term
| WHAT TYPE OF ARRHYTHMIAS IS BRETYLIUM USEFUL FOR? |
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Definition
| VENTRICULAR ARRHYTHMIAS, BUT NO EFECT ON ATRIAL TISSUE. |
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Term
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Definition
| INCREASES ACTION POTENTIAL DURATION IN NML AND ISCHEMIC CELLS. IT IS NO LONGER RECOMMENDED IN ACLS PROTOCOL. |
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Term
| WHAT ALL DOES AMIODARONE BLOCK? |
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Definition
| BLOCKS K, NA, AND CA CHANNELS ALONG WITH B RECEPTORS. IT DECREASES SA NODE AUTOMATOCITY, AV CONDUCTION VELOCITY AND PROLONGS AV NODE AND VENTRICULAR REFRACTORY PERIOD (BIG STOP SIGN FOR EVERYTHING CONDUCTION). |
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Term
| WHAT DOES AMIODARONE RESULT IN AN EKG? |
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Definition
| PROLONGS PRI, QRS, AND QTI |
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Term
| WHAT TYPE OF ARRHYTHMAIS CAN AMIODARONE BE USED TO TREAT? |
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Definition
| TREAT AND PREVENT RECEURRENCE OF ARIAL AND VENTRICULAR ARRHYTHMIAS |
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Term
| WHAT OTHER CLASS IS WITH AMIODARINE TO PREVENT ARRHYTHMIAS? |
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Definition
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Term
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Definition
| 150MG OVER 10 MINUTES, THEN INFUSION OF 1MG/MIN FOR 6 HRS OR 300MG DILUTED IN 20ML NS RAPID IV FOR PULSELESS VT OR VF. UP TO 5MG/KG FOR INITIAL LOADING DOSE. |
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Term
| WHAT IS THE HALG LIFE OF AMI? |
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Definition
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Term
| WHAT ARE 7 SIDE EFFECTS OF AMI? REMEMBER IT IS MOSTLY R/T TISSUE ACCUMULATION. AMIO JUST SITS IN TISSUE FOR EVEN YEARS AFTER ADMINISTRATION. |
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Definition
| PULMONARY FIBROSIS (DON'T USE HIGH FIO2), BRADYCARDIA, DECREASE TN(SOME SAY D/T A-BLOCKADE, SOME SAY D/T IV MIXTURE), THYORID TOXICITRY (BLOCKS CONVERSION OF T4 TO T3), PERIPHERAL NEUROPATHIES, OCULAR DEPOSITS, CYANOTIC- LOOKING SKIN |
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Term
| WHAT 4 MEDICATIONS HAVE DRUG INTERACTIONS WITH AMI? |
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Definition
| B-BLOCKERS, CCB, DIG (PROLONGED CARDIAC EFFECTS AND INCREASE R/O DIG TOXICITY), OPIOIDS ESP. FENTANYL= COMBINED BRADYCARDIC EFFECT. |
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Term
| IS AMI HIGHLY PROTEIN BOUND? |
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Definition
| YES UP TO 96&%. THIS CAN LEAD TO OTHER DRUG INTERACTIONS. |
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